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759 Mutation of this Cys to Ala or Ser largely inactivates the enzyme, which suggests that S -nitrosation may be a protective mechanism that preserves FolEIB activity under conditions of nitrosative stress

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This directly controls how cells migrate to repair sites, how they restructure damaged tissue scaffolding, and how quickly the cellular response to injury organizes itself

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Tandem MS Fragment-ion evidence helps distinguish sequence, terminal acetylation and possible derivatives