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Pretreatment with the GlyT1 inhibitors, sarcosine (competitive) and NFPS/ALX5407 (non-competitive), has been shown to promote neuroprotection in both ex vivo oxygenglucose deprivation (OGD) models and in vivo global cerebral ischemia models (Pinto et al

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Endothelial dysfunction in SAP Emerging evidence indicates that glycocalyx shedding precedes organ failure in SAP, serving as both a biomarker and a therapeutic target [22]

Z., Neumiller, J

Reduction in maintenance dose reflects that levels of cobalamin have been restored

The effects on EPSC frequency were associated with respective increases or decreases in intracellular glutamate