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Notably, MCT1 blockade in HCC cells promotes ferroptosis without observable alterations in GPX4 and FSP1 expression levels, while lactate uptake in melanoma cells via MCT1 elevates NADH, NADPH, GPX4 and FSP1

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Research involving this compound typically focuses on copper peptide signaling behavior, skin matrix pathway activity, collagen pathway research, extracellular matrix response, peptide-metal complex activity, cellular signaling, structure-function relationships, and measurable cellular response in experimental systems

T., Clemmons, D., Bellush, L