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Several potential carcinogenic mechanisms could play a role in LC development, including increased DNA methylation, mutations, and binding to the nicotinic acetylcholine receptor that could induce tumorigenesis, survival, and invasion [94]

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These cigarettes, as previously mentioned, are also presented in a compact format and have similar design elements

Upon exposure to nicotine, the release of granulocyte-macrophage colony-stimulating factor (GM-CSF) and the activation of Akt are also stimulated in airway epithelial cells (AECs) [93, 94]