& Hozo, I
pylori infection can mediate GC cells inflammation, epithelial-mesenchymal transition, migration, invasion, IL-8 secretion, and hummingbird-like changes in GC cells leading to disease progression [8,9,10,11]
[DOI] [PMC free article] [PubMed] [Google Scholar] [117].Zhou Y, Quan G, Wu Q
Existing evidence indicates that FSP1 localizes to plasma membranes and lipid droplets, and its anti-ferroptotic properties are due to its association with plasma membranes and not with lipid droplets [44]
the different populations analyzed (fetuses, newborns or infants)
Functional gene arrays-based analysis of fecal microbiomes in patients with liver cirrhosis