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357,361,371,372,373 Consistently, amygdalar axonal projections and neuronal activation are defective in Tbr1(+/) mice, but these defects are ameliorated by infusion of an NMDA receptor agonist (D-cycloserine)

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3 Genetic variants can contribute to gout development through numerous pathways, such as increasing urate production, reducing urate excretion or priming the immune systems inflammatory response to urate crystals