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Researchers utilize this blend to study complex dermatological models, specifically focusing on downregulating pro-inflammatory cytokines (like NF-B) in conditions resembling dermatitis, psoriasis, and compromised skin barriers

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This paradox arises from the inherent complexity of MS pathogenesis which we argue is driven by a convolution of relapsing/acute adaptive autoimmunity and chronic cytodegeneration (cyto-is used here rather than neuro-to underscore the point that myelin/oligodendrocytes are the likely primary targets of this process), including an elaborate innate immune response (Figure 1)

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