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This suggests that copper-binding and ROS production is not restricted to a small number of pathological proteins but may be a common property of intrinsically disordered proteins

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doi:10.3390/antiox12051094

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As for W303-1b/gsh1/pro1 or W303-1b/gsh1/proB, the overexpression of introduced Pro1 or ProB under the control of strong constitutive GAP promoter might allow a small amount of GSH in the host strains conferring tolerance to oxidative stress