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GS then catalyzes the second step, consuming another molecule of ATP to add glycine to -Glu-Cys, ultimately forming GSH

The study proposal was approved by Medical Ethics Committee of Tabriz University of Medical Sciences (IR.TBZMED.VCR.REC.1397.366)

In TP53 knockout mice, mitochondrial function and biomass are lost, whereas wild-type TP53 mice maintain mitochondrial biogenesis [84]

Metabolomics 6, 451465

Disruptions in this balance trigger ER stress responses that, if unresolved, contribute to diseases such as neurodegeneration and metabolic dysfunction 8