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As the primary factors for the reduced anticancer effect 1) the reduced delivery and uptake of the anticancer agent, 2) the need for the conversion of molecular oxygen (O 2 ) into reactive oxygen species (ROS), 3) the preferred therapeutic effect against highly proliferating cells, which is reduced under hypoxic conditions, or 4) adaptation of the cancer cells by promoting cell survival and upregulating certain resistance pathways, has been suggested 1,2,3,4,5

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Some researchers have argued that when this protective layer is compromised, the brain may become vulnerable to damage from chemicals, inflammatory cytokines, and immune cells

Even mitochondrial dynamics, which can be dictated by the redox environment, have been shown to play an integral role in dictating ROS-driven NSC cell fate decisions

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