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Treatment Am Fam Physician

These findings support the notion that differential redox capacity exists in different B cell subsets, and suggest that steady-state MZB require greater redox buffering capacity

A critical question remains: how can we ensure that ferroptosis is promoted exclusively in tumor cells while being inhibited in healthy ones

It binds to the antioxidant glutathione, enhancing the production of ROS, and leading to inflammation and mitochondrial damage [42, 43]

BK202010)

26 Recent literature has characterized GSH and other related metabolic enzymes as vital in protecting cells from ROS 27 through oxidation and reduction (redox) mechanisms