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This effect is mediated by modulating the expression of the anti-apoptotic protein B-cell lymphoma Extra Large (Bcl-XL) and the pro-apoptotic protein Bax, ultimately resulting in the programmed cell death of tumor cells via the cascade activation of the caspase-9/caspase-3 protease system (Malyarenko et al., 2019)

The role of oxidative stress in Parkinsons disease