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To further illustrate the observed in vivo effects of the BsNb-ncAA treated group, we analyzed infiltrating immune cells in the tumors of four groups of mice

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109 Alzheimers patients have changes in RAGE and LDL receptorrelated protein-1 concentrations on the hippocampus and cerebral cortex, thus indicating that A amyloid deposition secondary to BBB dysfunction is involved in the early pathogenesis of the disease