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Structural analysis demonstrates how FOXO4-DRI exploits the unique architecture of senescent cells, where FOXO4 latches onto p53 and prevents it from executing its normal pro-apoptotic function

Recent studies have leveraged the abnormal vascular structure of tumors, and the size advantage of nanoparticles (typically less than 200 nm), to achieve selective accumulation at prostate lesions via enhanced EPR

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