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Year 2020 Accessed 7 Aug 2023 Granata S, Zaza G, Simone S, Villani G, Latorre D, Pontrelli P, Carella M, Schena FP, Grandaliano G, Pertosa G (2009) Mitochondrial dysregulation and oxidative stress in patients with chronic kidney disease

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Further consideration for collecting data in this population is needed

Additional attention should be paid to studying the metabolism of SFN-nitriles and other GRP derivatives, and their roles in the body

Is it symptom-modifying, or is it truly disease modifying

Probably due to the binding preference of APAP to the active site of each P450 isomer, it is oxidized via two pathways to form the toxic intermediate N-acetyl-p-benzoquinone imine (NAPQI) and the non-toxic catechol metabolite 3-hydroxy-APAP (3-OH-APAP) (Chen et al., 1998)