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Elevated PARP-1 levels not only help against DNA damage, but are also exploited by EBV, as PARP1 acts as a barrier to productive lytic reactivation (229) and promotes the transition of EBV into the LMP1-induced latency state (230)

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228 Therefore, the co-administration of nicotinic acid and NAMPT inhibitors has been proposed as a new therapeutic strategy but only in NAPRT negative cancers

C., Tsai, H

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