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This isoform performs the same enzymatic reaction as ACSS2 in the mitochondria, primarily generating acetyl-CoA through fatty acid oxidation, which is further oxidized to CO [60]

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Proc Natl Acad Sci USA 111:1443614441 Zhang L, Luo H, Ni HM et al (2022) Ripk3 signaling regulates HSCs during stress and represses radiation-induced leukemia in mice

What exists instead is mechanistic plausibility: tirzepatide drives fat loss at rates faster than most patients have experienced before, and glutathione is the endogenous compound responsible for managing oxidative debris at the cellular level

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