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When your immune system fights infection, it generates reactive oxygen species that can damage healthy cells

This molecular mimicry can initiate and perpetuate autoimmune attacks on thyroid tissue, contributing to the progression of HT (Crotty, 2014)

Conclusions from 67 Cu uptake and release measurements performed in hypothalamic slices by the presence of action potential blocker tetrodotoxin [231, 232] or determination of depolarization-induced copper release from nerve endings by AAS [225] raised the concept of copper signalling in the brain

Hence, the development of effective novel treatments for AILI is critical

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