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doi: 10.7717/peerj.2503 75 StaceyN.ChojnackiA.NarayananA.ColeT.MurphyC

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Our findings propose another paradigm where ACE2 acts as a receptor-like protein, since pharmacological inhibition, as well as downregulation of ACE2 inhibited SARS-CoV-2 spike-induced inflammation, without any changes in ACE2 activity after exposure of human endothelial cells to the recombinant spike protein

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