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The end-result of SCD1 inhibition may, therefore, be an aggravation of NASH due to intracellular accumulation of cytotoxic saturated fatty acids [51, 62], placing the partitioning of saturated fatty acids to mono-unsaturated fatty acids as a protective factor in delaying NAFLD progression

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By contrast, ferrostatin-1-induced inhibition of ferroptosis decreased the average fluorescence intensity of ACSL4 in ECs (Fig

In addition to caloric reduction, recent preclinical studies have focused on chrono-nutritional regulation

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