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Br J Pharmacol 82:339347 Little HJ, Clark A, Watson WP (2000) Investigations into pharmacological antagonism of general anesthesia

Parallels are drawn with more traditional neurodegenerative disorders, where a progressive proteopathy with prion-like propagation of toxic misfolded species is now known to play a key role

It is not intended for human or animal consumption, clinical, diagnostic, therapeutic, dietary, or veterinary use

Further, it stimulates fibroblasts and increases nitric oxide production, aiding in tissue regeneration [3, 9]

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It was effectively applied either immediately (prophylactically) after paracetamol challenge or (therapeutically) administered after a 3 h period had elapsed (note, the maximum depletion of hepatic glutathione occurred 3 h after acute paracetamol dosing (37))