Melittin ameliorates CVB3-induced myocarditis via activation of the HDAC2-mediated GSK-3/Nrf2/ARE signaling pathway
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This pathway is involved in synapse formation, neurite outgrowth, and neuronal connectivity
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Neurons pretreated with Dihexa demonstrate significantly enhanced survival when exposed to reactive oxygen species, excitotoxic glutamate concentrations, or metabolic impairment compared to untreated controls
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