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10.1002/ajh.23317 Am
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B.DienzO.WargoM
Structure of the SARS-CoV-2 spike receptor-binding domain bound to the ACE2 receptor
Selfassembly of multinanozymes to mimic an intracellular antioxidant defense system
By further elucidating the pathogenic mechanism by which hepatic iron accumulation triggers ferroptosis through the c-Myc-Acsl4 regulatory axis, thereby accelerating the progression of metabolic-associated steatohepatitis (MASH), our research team identified a novel iron chelator, FOT1 (FerroTerminator1, formerly known as CN128), through drug screening, which can prevent and treat the onset and progression of MASH disease [117]
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