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Vitamin B12 deficiency and pernicious anaemia Peer reviewed by Dr Doug McKechnie, MRCGPLast updated by Dr Colin Tidy, MRCGPLast updated 5 Jul 2024 DownloadDownload Share Language Discussion Audio Version Add to preferred sources on Google In this series:AnaemiaDiets suitable for people with anaemiaFolic acid deficiencyIron-deficiency anaemiaMacrocytosis and macrocytic anaemia Vitamin B12 is essential for life

The ability of 31 to form a complex with LL5 and to promote the localization LL5 to the basal cortex indicates that integrins stabilize the plus ends of microtubules at the basal cell cortex through a laminin31LL5CLASPEB1microtubule linkage (Fig

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8 Despite methyltetrahydrofolate being the physiologically active form, some studies on patients with methyltetrahydrofolate receptor C677T genotype polymorphisms (a prominent genotype in autism) suggest that folinic acid supplementation may cause higher serum folate levels than similar doses of methyltetrahydrofolate

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(2017) Comparative bioavailability and utilization of particular forms of B(12) supplements with potential to mitigate B(12)-related genetic polymorphisms