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doi: 10.1016/j.theriogenology.2021.08.029

Remediation of Radiation-Induced Cognitive Dysfunction through Oral Administration of the Neuroprotective Compound NSI-189, 189(4), 345353

33 This highly inflammatory environment intensifies existing oxidative stress, further aggravating nerve damage

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Mechanistic analyses revealed that bacterial inactivation was associated to hydroxyl radical generation, and electrostatic interactions between positively charged N-CQDs and negatively charged bacterial membranes, leading to morphological changes and cell wall damage [133]