They actively disrupt the stoichiometry of photosynthetic proteins, degrade PSII, and change the redox state of the plastoquinone pool, resulting in the excessive production and accumulation of ROS, which eventually triggers a cell death that resembles the effector-triggered HR that limits the growth of pathogens 17,23,24,59,60,61
Evidence quality remains limited, and results vary significantly between individuals
Implications for Research, Policy, and Practice
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