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VPA treatment, at the progenitor stage of hippocampal treatment, led to robust inhibition of cell proliferation and stimulation of neuronal differentiation due to an increase in expression of proneural transcription factor

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In turn, inactive MnSOD drives oxidative stress while promoting prooxidative proliferation and initiating a downstream effect on AKT activation, potentiating the survival of fibroid cells in a prooxidative environment [55]